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Trenbolone binds to androgen receptors (ARs) with approximately three times the affinity of testosterone and has been shown to augment skeletal muscle mass and bone growth and reduce adiposityand body fat and increase lean body mass [14]. Since the binding of testosterone and trenbolone, to other ligands of AR (i.e. ARB and ARCC) is more tightly regulated than AR binding to testosterone [15], we tested whether a single dose of an AR blocker would inhibit testosterone binding to the ARs Trenbolone and Testerone.
Materials and Methods
Subjects.
Subjects were recruited from the local area. Of the initial 25 participants, 26 (17 women and 7 men) agreed to participate (age range 23.6 ± 1.8 years; body mass index: 27.6 ± 2.5 kg/m2; mean ± SD age) including 4 women and 2 men who were either not willing or unable to continue. After informed informed consent and an oral informed consent form, each subject provided a written informed consent form before receiving the drug.
Drug Delivery Systems.
Dihydrotestosterone (DHT) and methenolone acetate (MA) were purchased from Sigma. Testosterone enanthate (TGA) was purchased from Sigma. Trenbolone and testerone were dissolved in 100% distilled water. Methyldopa was purchased from Tocris, Glaxo Smith Kline, and is approved for the use of medical residents in several countries. All of the other drugs were obtained from Sigma.
Study Design.
Subjects completed 2 d of randomization, which began on Day 1.
After receiving their assigned doses of testosterone and/or DHT, subjects participated in 2-h tests of basal and free- testosterone levels. After 2 h of baseline tests (day 1), subjects received a single dose of the AR blocker, the testosterone antagonist, or both testosterone and the antiandrogen, either alone or together. Before and after receiving each dose, and/or before and after 3 months of dosing, subjects underwent laboratory testing including blood, urine and oral glucose tolerance tests (OGTTs), which were repeated each day in each subject after each dose of drug. After each drug treatment, subjects were given an oral glucose tolerance test. The subjects were also given 1 d of washout before conducting all subsequent tests.
Statistical Analysis
Baseline data were compared between groups using a paired Wilcoxon signed rank test. Following each baseline testing day, baseline data were compared between groups using a nonparametric repeated measures ANOVA (one-
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